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Modern Approaches in Pharmacy & Pharmaceutics Sciences is a one of the ideal journal in pharmaceutical journals, this journal focuses to deliver most polish and reliable wellspring of information on the revelations and current developments in the method of Research, review, case reports, communications, etc. in numerous aspects of the field of Pharmaceutical and Pharmacological studies and getting them freely available through electronically with no limitations. The aim of the journal is to work together to develop novel therapies and approaches to patient care. To accomplish this, we need your exceptional research and innovation to the world promptly in the pharmacy journal. Our journal is a peer-reviewed international journal that offers active pharmacists in-depth worthwhile assessments in Pharmacy informatics, Pharmaceutics, Pharmacokinetics, Medicinal Chemistry, Pharmacognosy, Pharmacology, Pharmacodynamics, Toxicology, Pharmacogenomics, Pharmaceutical Analysis, Drug interactions and other pharmacy practice topics.
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Certificate of Standardized Training for Residents (China) 1998.12-2001.12 Qilu Hospital of Shandong University Certificate of Educational Commission for Foreign Medical Graduates （ECFMG, the United States）2011 Licensed Family Doctor 2023 Licensed Physician in Gastroenterology 1998 Senior Public Dietitian (Grade III, China) 2022 ICH-GCP Certificate (GCP certificate issued by NIDA, USA) 2022.09.06 Zhengzhou Sixth People’s Hospital (i.e. Henan Provincial Infectious Disease Hospital)(National tertiary A hospital), Professor, Head of International Healthcare Unit, Associate director of clinical laboratory, Research Officer of Hepatology Department 2015.09 to now The third affiliated Hospital of Xinxiang Medical University (Tertiary Hospital), Department of Gastroenterology and Hepatology, 2014.07～2015.08 Qianfoshan Hospital of Shandong University (Tertiary A hospital), Department of Gastroenterology & Hepatology, attending physician, 2013.07～2014.07 Texas University, Health Science Center at San Antonio, Post doctorate/Research associate, 2006.07～2013.07 Qilu Hospital of Shandong University (National tertiary A hospital), Department of Gastroenterology and Hepatology, attending physician, 1998.07～2008.03
Liver Disease, Molecular Biology etc.
Dr. Feng Jin, senior engineer of biotechnology, is currently the deputy general manager of Shenzhen NewDEL Biotechnology Co., Ltd. and the director of the research and development department of Guangdong Province Small Molecular New Drug Innovation Center. Dr. Feng Jin graduated from Tsinghua University in July 2012 with a doctors degree in chemistry. After that, he has been engaged in the discovery and development of innovative drugs in Shenzhen Neptunus Pharmaceutical Research Institute. Dr. Jin Fengs main research direction is the discovery and development of targeted anti-tumor drugs, and has published more than 20 papers in relevant international journals. In 2018, as a visiting scholar, he spent one year in the laboratory of Professor Richard Zare of Stanford University, engaged in the research of drug molecular design and microdroplet synthesis. After returning to China in 2019, Dr. Jin Feng joined AI pharmaceutical company—Shenzhen Zhiyao Information Technology Co., Ltd. as Chief Scientific Officer. In 2021, Dr. Jin Feng joined Guangdong Small Molecular New Drug Innovation Center and Shenzhen NewDEL Biotechnology Co., Ltd
Biotechnology
Professional Experience: Cardiologist, Department of Cardiology, Aerospace Center Hospital Member of the Cardiovascular Branch of the Chinese Society of Pathophysiology Member of Cardiovascular Branch of China Geriatric Health Association Academic Achievements / Publications: (1) National Natural Science Foundation of China, general project, 82170243, new mechanism of regulation and activation of cardiac fibroblasts: endogenous sulfur dioxide dependent WT1 sulfonation modification, from January 1, 2022 to December 31, 2025, under research, participating (2) National Natural Science Foundation of the Peoples Republic of China, Youth Science Foundation Project, 82000289, Hydrogen sulfide inhibits the activation of atrial fibroblasts Smo-Gli and antagonizes the atrial fibrosis of atrial fibrillation, from January 1, 2021 to December 31, 2023, under research, presiding (3) National Natural Science Foundation of China, general project, 81770278, SRF/MRTF-A pathway mediates the regulatory effect and mechanism of endogenous sulfur dioxide on the phenotypic transformation of vascular smooth muscle cells during neointima formation, from January 1, 2018 to December 31, 2021, conclusion, participation. (4) Rui Wang; Yong Fen Qi; Chun Hua Ding; Da Zhang ; Sulfur-containing amino acids and their metabolites in atrial fibrosis, Front Pharmacol., 2022, 13(1063241) (5) Wence Shi; Wenchang Zhang; Da Zhang; Guojie Ye; Chunhua Ding ; Mortality and Clinical Pre dictors After Percutaneous Mitral Valve Repair for Secondary Mitral Regurgitation: A Systematic R eview and Meta-Regression Analysis, Front Cardiovasc Med, 2022, 9(918712) (6) Shi WC; Zhang WC; Zhang D; Ren G; Wang PF; Gao LH; Chen HN; Ding CH ; Comparison of the e ffect of glucose-lowering agents on the risk of atrial fibrillation: A network meta-analysis., He art Rhythm, 2021, 18(7): 1090-1096 (7) Zhang D; Wang XL; Chen SY; Chen S; Yu W; Liu X; Yang GS; Tao YH; Tang XJ; Bu DF; Zhang H; Kong W; Tang CS; Huang YQ; Du JB; Jin HF ; Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation, Clinical Science, 2019, 133: 2045-2059 (8) Wang XL; Wang Y; Zhang LL; Zhang D; Bai L; Kong W; Huang YQ; Tang CS; Du JB; Jin HF ; L-c ystathionine protects against homocysteine-induced mitochondria-dependent apoptosis of vascular e ndothelial cells, Oxidative Medicine and Cellular Longevity, 2019, 2019(1253289) (9) Zhang D; Wang XL; Tian XY; Zhang LL; Yang GS; Tao YH; Liang C; Li K; Yu XQ; Tang XJ; Tang CS; Zhou J; Kong W; Du JB; Huang YQ; Jin HF. The increased endogenous sulfur Dioxide acts as a compensatory Mechanism for the Downregulated endogenous hydrogen sulfide Pathway in the endothelial cell inflammation. Frontiers in Immunology. 2018,9:882 (10) Feng SS; Chen SY; Yu W; Zhang D; Zhang CY; Tang CS; Du JB; Jin HF. H2S inhibits pulmonary arterial endothelial cell inflammation in rats with monocrotaline-induced pulmonary hypertension Laboratory Investigation, 2017, 97: 268-278 (11) Zhang D; Du JB; Tang CS; Huang YQ; Jin HF. H2S-induced sulfhydration: biological function and detection methodology. Frontiers in Pharmacology, 2017, 8(608)
Professional Experience: Cardiologist, Department of Cardiology, Aerospace Center Hospital Member of the Cardiovascular Branch of the Chinese Society of Pathophysiology Member of Cardiovascular Branch of China Geriatric Health Association Academic Achievements / Publications: (1) National Natural Science Foundation of China, general project, 82170243, new mechanism of regulation and activation of cardiac fibroblasts: endogenous sulfur dioxide dependent WT1 sulfonation modification, from January 1, 2022 to December 31, 2025, under research, participating (2) National Natural Science Foundation of the Peoples Republic of China, Youth Science Foundation Project, 82000289, Hydrogen sulfide inhibits the activation of atrial fibroblasts Smo-Gli and antagonizes the atrial fibrosis of atrial fibrillation, from January 1, 2021 to December 31, 2023, under research, presiding (3) National Natural Science Foundation of China, general project, 81770278, SRF/MRTF-A pathway mediates the regulatory effect and mechanism of endogenous sulfur dioxide on the phenotypic transformation of vascular smooth muscle cells during neointima formation, from January 1, 2018 to December 31, 2021, conclusion, participation. (4) Rui Wang; Yong Fen Qi; Chun Hua Ding; Da Zhang ; Sulfur-containing amino acids and their metabolites in atrial fibrosis, Front Pharmacol., 2022, 13(1063241) (5) Wence Shi; Wenchang Zhang; Da Zhang; Guojie Ye; Chunhua Ding ; Mortality and Clinical Pre dictors After Percutaneous Mitral Valve Repair for Secondary Mitral Regurgitation: A Systematic R eview and Meta-Regression Analysis, Front Cardiovasc Med, 2022, 9(918712) (6) Shi WC; Zhang WC; Zhang D; Ren G; Wang PF; Gao LH; Chen HN; Ding CH ; Comparison of the e ffect of glucose-lowering agents on the risk of atrial fibrillation: A network meta-analysis., He art Rhythm, 2021, 18(7): 1090-1096 (7) Zhang D; Wang XL; Chen SY; Chen S; Yu W; Liu X; Yang GS; Tao YH; Tang XJ; Bu DF; Zhang H; Kong W; Tang CS; Huang YQ; Du JB; Jin HF ; Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation, Clinical Science, 2019, 133: 2045-2059 (8) Wang XL; Wang Y; Zhang LL; Zhang D; Bai L; Kong W; Huang YQ; Tang CS; Du JB; Jin HF ; L-c ystathionine protects against homocysteine-induced mitochondria-dependent apoptosis of vascular e ndothelial cells, Oxidative Medicine and Cellular Longevity, 2019, 2019(1253289) (9) Zhang D; Wang XL; Tian XY; Zhang LL; Yang GS; Tao YH; Liang C; Li K; Yu XQ; Tang XJ; Tang CS; Zhou J; Kong W; Du JB; Huang YQ; Jin HF. The increased endogenous sulfur Dioxide acts as a compensatory Mechanism for the Downregulated endogenous hydrogen sulfide Pathway in the endothelial cell inflammation. Frontiers in Immunology. 2018,9:882 (10) Feng SS; Chen SY; Yu W; Zhang D; Zhang CY; Tang CS; Du JB; Jin HF. H2S inhibits pulmonary arterial endothelial cell inflammation in rats with monocrotaline-induced pulmonary hypertension Laboratory Investigation, 2017, 97: 268-278 (11) Zhang D; Du JB; Tang CS; Huang YQ; Jin HF. H2S-induced sulfhydration: biological function and detection methodology. Frontiers in Pharmacology, 2017, 8(608)
Professional Experience: Cardiologist, Department of Cardiology, Aerospace Center Hospital Member of the Cardiovascular Branch of the Chinese Society of Pathophysiology Member of Cardiovascular Branch of China Geriatric Health Association Academic Achievements / Publications: (1) National Natural Science Foundation of China, general project, 82170243, new mechanism of regulation and activation of cardiac fibroblasts: endogenous sulfur dioxide dependent WT1 sulfonation modification, from January 1, 2022 to December 31, 2025, under research, participating (2) National Natural Science Foundation of the Peoples Republic of China, Youth Science Foundation Project, 82000289, Hydrogen sulfide inhibits the activation of atrial fibroblasts Smo-Gli and antagonizes the atrial fibrosis of atrial fibrillation, from January 1, 2021 to December 31, 2023, under research, presiding (3) National Natural Science Foundation of China, general project, 81770278, SRF/MRTF-A pathway mediates the regulatory effect and mechanism of endogenous sulfur dioxide on the phenotypic transformation of vascular smooth muscle cells during neointima formation, from January 1, 2018 to December 31, 2021, conclusion, participation. (4) Rui Wang; Yong Fen Qi; Chun Hua Ding; Da Zhang ; Sulfur-containing amino acids and their metabolites in atrial fibrosis, Front Pharmacol., 2022, 13(1063241) (5) Wence Shi; Wenchang Zhang; Da Zhang; Guojie Ye; Chunhua Ding ; Mortality and Clinical Pre dictors After Percutaneous Mitral Valve Repair for Secondary Mitral Regurgitation: A Systematic R eview and Meta-Regression Analysis, Front Cardiovasc Med, 2022, 9(918712) (6) Shi WC; Zhang WC; Zhang D; Ren G; Wang PF; Gao LH; Chen HN; Ding CH ; Comparison of the e ffect of glucose-lowering agents on the risk of atrial fibrillation: A network meta-analysis., He art Rhythm, 2021, 18(7): 1090-1096 (7) Zhang D; Wang XL; Chen SY; Chen S; Yu W; Liu X; Yang GS; Tao YH; Tang XJ; Bu DF; Zhang H; Kong W; Tang CS; Huang YQ; Du JB; Jin HF ; Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation, Clinical Science, 2019, 133: 2045-2059 (8) Wang XL; Wang Y; Zhang LL; Zhang D; Bai L; Kong W; Huang YQ; Tang CS; Du JB; Jin HF ; L-c ystathionine protects against homocysteine-induced mitochondria-dependent apoptosis of vascular e ndothelial cells, Oxidative Medicine and Cellular Longevity, 2019, 2019(1253289) (9) Zhang D; Wang XL; Tian XY; Zhang LL; Yang GS; Tao YH; Liang C; Li K; Yu XQ; Tang XJ; Tang CS; Zhou J; Kong W; Du JB; Huang YQ; Jin HF. The increased endogenous sulfur Dioxide acts as a compensatory Mechanism for the Downregulated endogenous hydrogen sulfide Pathway in the endothelial cell inflammation. Frontiers in Immunology. 2018,9:882 (10) Feng SS; Chen SY; Yu W; Zhang D; Zhang CY; Tang CS; Du JB; Jin HF. H2S inhibits pulmonary arterial endothelial cell inflammation in rats with monocrotaline-induced pulmonary hypertension Laboratory Investigation, 2017, 97: 268-278 (11) Zhang D; Du JB; Tang CS; Huang YQ; Jin HF. H2S-induced sulfhydration: biological function and detection methodology. Frontiers in Pharmacology, 2017, 8(608)
Professional Experience: Cardiologist, Department of Cardiology, Aerospace Center Hospital Member of the Cardiovascular Branch of the Chinese Society of Pathophysiology Member of Cardiovascular Branch of China Geriatric Health Association Academic Achievements / Publications: (1) National Natural Science Foundation of China, general project, 82170243, new mechanism of regulation and activation of cardiac fibroblasts: endogenous sulfur dioxide dependent WT1 sulfonation modification, from January 1, 2022 to December 31, 2025, under research, participating (2) National Natural Science Foundation of the Peoples Republic of China, Youth Science Foundation Project, 82000289, Hydrogen sulfide inhibits the activation of atrial fibroblasts Smo-Gli and antagonizes the atrial fibrosis of atrial fibrillation, from January 1, 2021 to December 31, 2023, under research, presiding (3) National Natural Science Foundation of China, general project, 81770278, SRF/MRTF-A pathway mediates the regulatory effect and mechanism of endogenous sulfur dioxide on the phenotypic transformation of vascular smooth muscle cells during neointima formation, from January 1, 2018 to December 31, 2021, conclusion, participation. (4) Rui Wang; Yong Fen Qi; Chun Hua Ding; Da Zhang ; Sulfur-containing amino acids and their metabolites in atrial fibrosis, Front Pharmacol., 2022, 13(1063241) (5) Wence Shi; Wenchang Zhang; Da Zhang; Guojie Ye; Chunhua Ding ; Mortality and Clinical Pre dictors After Percutaneous Mitral Valve Repair for Secondary Mitral Regurgitation: A Systematic R eview and Meta-Regression Analysis, Front Cardiovasc Med, 2022, 9(918712) (6) Shi WC; Zhang WC; Zhang D; Ren G; Wang PF; Gao LH; Chen HN; Ding CH ; Comparison of the e ffect of glucose-lowering agents on the risk of atrial fibrillation: A network meta-analysis., He art Rhythm, 2021, 18(7): 1090-1096 (7) Zhang D; Wang XL; Chen SY; Chen S; Yu W; Liu X; Yang GS; Tao YH; Tang XJ; Bu DF; Zhang H; Kong W; Tang CS; Huang YQ; Du JB; Jin HF ; Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation, Clinical Science, 2019, 133: 2045-2059 (8) Wang XL; Wang Y; Zhang LL; Zhang D; Bai L; Kong W; Huang YQ; Tang CS; Du JB; Jin HF ; L-c ystathionine protects against homocysteine-induced mitochondria-dependent apoptosis of vascular e ndothelial cells, Oxidative Medicine and Cellular Longevity, 2019, 2019(1253289) (9) Zhang D; Wang XL; Tian XY; Zhang LL; Yang GS; Tao YH; Liang C; Li K; Yu XQ; Tang XJ; Tang CS; Zhou J; Kong W; Du JB; Huang YQ; Jin HF. The increased endogenous sulfur Dioxide acts as a compensatory Mechanism for the Downregulated endogenous hydrogen sulfide Pathway in the endothelial cell inflammation. Frontiers in Immunology. 2018,9:882 (10) Feng SS; Chen SY; Yu W; Zhang D; Zhang CY; Tang CS; Du JB; Jin HF. H2S inhibits pulmonary arterial endothelial cell inflammation in rats with monocrotaline-induced pulmonary hypertension Laboratory Investigation, 2017, 97: 268-278 (11) Zhang D; Du JB; Tang CS; Huang YQ; Jin HF. H2S-induced sulfhydration: biological function and detection methodology. Frontiers in Pharmacology, 2017, 8(608)
Professional Experience: Cardiologist, Department of Cardiology, Aerospace Center Hospital Member of the Cardiovascular Branch of the Chinese Society of Pathophysiology Member of Cardiovascular Branch of China Geriatric Health Association Academic Achievements / Publications: (1) National Natural Science Foundation of China, general project, 82170243, new mechanism of regulation and activation of cardiac fibroblasts: endogenous sulfur dioxide dependent WT1 sulfonation modification, from January 1, 2022 to December 31, 2025, under research, participating (2) National Natural Science Foundation of the Peoples Republic of China, Youth Science Foundation Project, 82000289, Hydrogen sulfide inhibits the activation of atrial fibroblasts Smo-Gli and antagonizes the atrial fibrosis of atrial fibrillation, from January 1, 2021 to December 31, 2023, under research, presiding (3) National Natural Science Foundation of China, general project, 81770278, SRF/MRTF-A pathway mediates the regulatory effect and mechanism of endogenous sulfur dioxide on the phenotypic transformation of vascular smooth muscle cells during neointima formation, from January 1, 2018 to December 31, 2021, conclusion, participation. (4) Rui Wang; Yong Fen Qi; Chun Hua Ding; Da Zhang ; Sulfur-containing amino acids and their metabolites in atrial fibrosis, Front Pharmacol., 2022, 13(1063241) (5) Wence Shi; Wenchang Zhang; Da Zhang; Guojie Ye; Chunhua Ding ; Mortality and Clinical Pre dictors After Percutaneous Mitral Valve Repair for Secondary Mitral Regurgitation: A Systematic R eview and Meta-Regression Analysis, Front Cardiovasc Med, 2022, 9(918712) (6) Shi WC; Zhang WC; Zhang D; Ren G; Wang PF; Gao LH; Chen HN; Ding CH ; Comparison of the e ffect of glucose-lowering agents on the risk of atrial fibrillation: A network meta-analysis., He art Rhythm, 2021, 18(7): 1090-1096 (7) Zhang D; Wang XL; Chen SY; Chen S; Yu W; Liu X; Yang GS; Tao YH; Tang XJ; Bu DF; Zhang H; Kong W; Tang CS; Huang YQ; Du JB; Jin HF ; Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation, Clinical Science, 2019, 133: 2045-2059 (8) Wang XL; Wang Y; Zhang LL; Zhang D; Bai L; Kong W; Huang YQ; Tang CS; Du JB; Jin HF ; L-c ystathionine protects against homocysteine-induced mitochondria-dependent apoptosis of vascular e ndothelial cells, Oxidative Medicine and Cellular Longevity, 2019, 2019(1253289) (9) Zhang D; Wang XL; Tian XY; Zhang LL; Yang GS; Tao YH; Liang C; Li K; Yu XQ; Tang XJ; Tang CS; Zhou J; Kong W; Du JB; Huang YQ; Jin HF. The increased endogenous sulfur Dioxide acts as a compensatory Mechanism for the Downregulated endogenous hydrogen sulfide Pathway in the endothelial cell inflammation. Frontiers in Immunology. 2018,9:882 (10) Feng SS; Chen SY; Yu W; Zhang D; Zhang CY; Tang CS; Du JB; Jin HF. H2S inhibits pulmonary arterial endothelial cell inflammation in rats with monocrotaline-induced pulmonary hypertension Laboratory Investigation, 2017, 97: 268-278 (11) Zhang D; Du JB; Tang CS; Huang YQ; Jin HF. H2S-induced sulfhydration: biological function and detection methodology. Frontiers in Pharmacology, 2017, 8(608)
Bio chemistry
University of Texas Medical Branch, USA
Department of Criminal Justice
Liberty University, USA
Department of Psychiatry
University of Kentucky, USA
Department of Medicine
Gally International Biomedical Research & Consulting LLC, USA
Department of Urbanisation and Agricultural
Montreal university, USA
Oral & Maxillofacial Pathology
New York University, USA
Gastroenterology and Hepatology
University of Alabama, UK
Department of Medicine
Universities of Bradford, UK
Oncology
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Radiation Chemistry
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Analytical Chemistry
Wentworth Institute of Technology, USA
Minimally Invasive Surgery
Mercer University school of Medicine, USA
Pediatric Dentistry
University of Athens , Greece