Trainini Jorge MD, PhD*1,Lowenstein Jorge MD2, Beraudo Mario MD3, Wernicke Mario MD4, Jesús Valle, PhD5, Lowenstein-Haber Diego MD2, Labrada Luis MD6, Trainini Alejandro MD1,3 and Bastarrica María Elena MD3
Received:April 08, 2021; Published: April 20, 2021
Corresponding author: Trainini Jorge MD, PhD, Department of Cardiac Surgery, Hospital Presidente Perón, Buenos Aires, Argentina
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The role of the vortex in cardiac remodeling should be understood as an etiopathogenic factor in the myocardial wall with its consequent dilation and not as a cause of this wall’s alteration. It is not necessary to consider the random molecular behavior in the intraventricular vortex as maximum unpredictability. Randomness becomes uniformity. For example, in a balloon, particles move in all directions but exert the same pressure. This is analogous to the left ventricle: molecules collide against each other as a dense and anarchic crowd. A fluid applies pressure on the surface with which it makes contact, but if the chaotic elements become ordered, we have regularity. A milliliter of blood contains around one hundred trillion particles. It is impossible to combine their equations. Probability in statistics was used to find regularity in global and average behavior. The random state of molecular chaos (turbulence) that occurs in the intraventricular vortex alters the wall. Remodeling leading to volume overload starts when 20% of the ventricular mass is compromised. With increased volume there is more sphericity and vice versa. Thus, fluid becomes a sculptor of the ventricular wall through the velocity of fluid particles colliding against its walls.
Keywords: Intraventricular vortex; Dilated cardiomyopathy; Myocardial torsion
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